One Drug Halted, Another Advances

Novartis halted one experimental mesothelioma drug, but a competitor's TEAD inhibitor shows strong results in clinical trials.

Key Facts
Novartis IAG933 discontinued—reason undisclosed
VT3989 now the leading TEAD inhibitor
VT3989: 32% response, 86% disease control, 40-week PFS
Phase 3 trials planned for 2026

Novartis has quietly discontinued development of IAG933, its TEAD inhibitor being studied for mesothelioma. The decision leaves Vivace Therapeutics’ VT3989 as the leading drug in this promising class, which targets a key vulnerability in mesothelioma cells.

What Are TEAD Inhibitors?

The Hippo Pathway

TEAD inhibitors target a cellular pathway critical to many mesotheliomas:

Pathway ComponentRoleIn Mesothelioma
Hippo pathwayControls cell growthOften disrupted
NF2/MerlinTumor suppressorLost in 40-50% of cases
YAP/TAZGrowth signalsOveractive when NF2 lost
TEADFinal effectorDrug target

When the tumor suppressor NF2 is lost (common in mesothelioma), TEAD proteins drive uncontrolled cell growth. Blocking TEAD can stop this growth.

Why TEAD Is Attractive

AdvantageExplanation
Mesothelioma-specificHigh rates of pathway disruption
Novel mechanismDifferent from chemo and immunotherapy
Oral dosingConvenient for patients
Clear biomarkerNF2 status may predict response

The Novartis Discontinuation

What Happened

TimelineEvent
2020sNovartis develops IAG933
2024Phase 1/2 trials underway
Late 2025Development quietly halted
ReasonNot publicly disclosed

Novartis has not explained why IAG933 was discontinued. Possible reasons include:

  • Insufficient efficacy
  • Safety concerns
  • Strategic portfolio decisions
  • Competition from other programs

Industry Context

Novartis joins several companies that have struggled with TEAD inhibitors:

CompanyDrugStatus
NovartisIAG933Discontinued
IkenaIK930Limited progress
VivaceVT3989Advancing to Phase 3

The challenges highlight the difficulty of developing TEAD inhibitors, but also make VT3989’s success more significant.

One Drug's Failure Doesn't Doom a Class

Multiple TEAD inhibitor programs have struggled, but VT3989’s success validates the target. For patients seeking TEAD inhibitor access, ask your oncologist about VT3989 clinical trials and consider NF2 testing—this biomarker may predict response.

VT3989: Now the Leader

Strong Clinical Data

With Novartis out, VT3989 from Vivace Therapeutics is now the most advanced TEAD inhibitor for mesothelioma:

VT3989 ResultsData
Overall response rate32%
Disease control rate86%
Median PFS40 weeks (vs 15 weeks historical)
FDA designationFast Track
Next stepPhase 3 trial planned

Why VT3989 May Have Succeeded

FactorPossible Advantage
Drug designDifferent binding characteristics
Patient selectionFocused on NF2-mutant tumors
Dosing strategyOptimized regimen
Company focusMesothelioma as priority indication

What This Means for Patients

Current Treatment Landscape

TreatmentSettingStatus
ChemotherapyFirst-lineStandard of care
Opdivo + YervoyFirst-lineFDA approved
Nivolumab aloneRelapsedCONFIRM trial data
TEAD inhibitorsRelapsedIn development

TEAD inhibitors represent a potential new option for patients who have exhausted standard treatments.

Timeline Expectations

MilestoneExpected Timeframe
VT3989 Phase 3 starts2026
Phase 3 results2028-2029
Potential FDA approval2029-2030

Patients seeking TEAD inhibitor access should ask about clinical trials.

Finding TEAD Inhibitor Trials

  1. Ask your oncologist about VT3989 and other TEAD trials
  2. Search ClinicalTrials.gov for “TEAD mesothelioma”
  3. Consider NF2 testing: May help determine eligibility
  4. Contact Vivace Therapeutics: For trial information

The Broader Drug Development Lesson

High Failure Rates

The IAG933 discontinuation illustrates drug development challenges:

StageHistorical Success Rate
Phase 1 to approval~10%
Phase 2 to approval~30%
Phase 3 to approval~60%

Many promising drugs fail. The key is having multiple options in development.

Mesothelioma Pipeline Status

Drug ClassDrugs in Development
Checkpoint inhibitorsMultiple approved and in trials
TEAD inhibitorsVT3989 leading
Arginine depletionPegargiminase
Bispecific antibodiesBNT327 and others
Targeted therapiesVarious

The pipeline is more robust than ever, offering hope despite individual setbacks.

Key Takeaways

  1. One drug’s failure doesn’t doom a class: VT3989 continues to show promise
  2. Mesothelioma research is advancing: Multiple novel approaches in development
  3. Biomarkers matter: NF2 status may predict TEAD inhibitor response
  4. Clinical trials are essential: Patients can access new drugs through trials
  5. Patience required: Drug development takes years
What happened to Novartis IAG933?

Novartis quietly discontinued development of IAG933 in late 2025. The company has not disclosed the reason—it could be insufficient efficacy, safety concerns, strategic decisions, or competition from other programs. This leaves VT3989 as the leading TEAD inhibitor for mesothelioma.

What are TEAD inhibitors?

TEAD inhibitors target a key pathway in mesothelioma. When the tumor suppressor NF2 is lost (40-50% of cases), TEAD proteins drive uncontrolled cell growth. Blocking TEAD can stop this growth. The approach is mesothelioma-specific, oral, and has a clear biomarker (NF2 status).

How is VT3989 performing?

VT3989 has shown 32% response rate, 86% disease control, and 40-week median progression-free survival (vs 15 weeks historical). It has received FDA Fast Track designation and is advancing to Phase 3 trials starting in 2026, with potential approval around 2029-2030.

How can I access TEAD inhibitor trials?

Ask your oncologist about VT3989 trials, search ClinicalTrials.gov for “TEAD mesothelioma,” consider NF2 testing to determine eligibility, and contact Vivace Therapeutics directly for trial information.